portal area

Portal area was not typical .

肝门管区不典型。

Apoptotic cells distributed at the periphery of central vein and that of portal area .

凋亡细胞分布于中央静脉周围和门管区周围。

42 ( 79.25 % ) of inflammatory cells infiltration in portal area ;

汇管区炎细胞浸润42例次（79.25%）。

IL-17 mainly located in portal area and was associated with fibrosis extent closely .

肝组织IL-17染色位于汇管区，与纤维化高度相关；

Fibroblasts mainly distribute in the portal area and are the major type cells in liver stroma .

成纤维母细胞是肝脏基质细胞中的主要细胞类型之一，主要分布在肝脏的门脉部位。

Liver cells granular degeneration and fatty degeneration , vessels of portal area engorgement ;

肝细胞发生颗粒和脂肪变性，汇管区血管充血；

Liver cell degeneration , necrosis and portal area inflammation were also reduced in 3 cases ( P < 0.05 ) .

7例患者肝细胞变性坏死、汇管区炎症减轻（P<0.05）。

Focal necrosis , infiltration of inflammatory cells in portal area and multinucleate giant cell in liver ;

肝脏局灶性坏死，门管区炎性细胞浸润，出现多核巨细胞；

Results By day2 after partial hepatectomy , OC began to proliferate around the portal area .

结果肝部分切除后第2天，卵圆细胞开始向门静脉周围区域增殖。

But during the hepatectomy near the first , second and third hepatic portal area , the hemostatic ways have their own specialty .

但靠近第一、二、三肝门的肝切除，止血措施有各自的特殊性。

The pathological features of autoimmune hepatitis is inflammatory cell infiltration in the portal area and invading to the surrounding liver parenchyma .

自身免疫性肝炎的病理特征为大量炎性细胞浸润于汇管区，并向周围的肝实质侵犯。

In the control group , the structure of liver lobule was normal and a few of collagen fibers located in portal area and central vein .

对照组肝小叶结构和肝细胞状态正常，仅在中央静脉及汇管区可见少量胶原纤维。

In the Ganshu group , fibrous connective tissue proliferation to form short intervals can be seen in the portal area and surrounding .

肝俞组大鼠在汇管区及周围见纤维结缔组织增生，可见短间隔形成。

In this group 15 cases occurred by cholecystectomy ( 15 / 18 83.3 % ), and mostly were injured in the biliary duct of portal area .

本组15例（15/1883.3%）由胆囊手术操作不当，主要发生于汇管区胆管。

Portal area is not typical , but still can find out the interlobular artery , interlobular vein and interlobular bile duct in portal area .

肝门管区不典型，仍可找到伴行的小叶间动脉、小叶间静脉和小叶间胆管。

The tissue structure of the liver mainly included hepatic lobule and portal area . Central vein , hepatocyte tube and hepatic sinusoid were found in hepatic lobule .

肝脏的组织结构由肝小叶和门管区构成，肝小叶由中央静脉、肝细胞管和肝窦组成，门管区包括小叶间胆管、小叶间动脉和小叶间静脉，还有淋巴组织分布。

And the expression of Bcl-2 in liver fibrosis rats tissue was obviously higher than the normal liver tissue , with the main expression in the fibrous septae and portal area .

bcl-2在肝纤维化大鼠肝组织中的表达明显高于正常肝组织，且主要表达在纤维间隔、汇管区等组织中。

Hepatic cord arranged in disorder in middle dose ethanol group . Connective tissue hyperplasia in the liver portal area and spotty necrosis can be observed in high dose ethanol group .

乙醇中剂量组肝索紊乱；高剂量组门管区结缔组织增生，肝实质区可见点状坏死。

Results MMP-2 , MT-MMP-2 mRNA and related antigens were expressed in mesenchymal cells and parts of hepatocytes besides active pathological changes , especially in the fibrous septum and portal area .

结果MMP2、MTMMP2mRNA及相关抗原在间质细胞和部分肝细胞中表达，以纤维间隔及汇管区最为明显。

CD34 and c-kit were detected at the 8th week in some ductal plate cells and a few mononuclear cells in the hepatic cords / mesenchymal tissue of portal area ;

8周龄胚胎肝胆管板中开始可见CD34和c-kit阳性细胞，肝索和门静脉附近间充质内也可见少数表达CD34和c-kit的单个核细胞；

Cell-specific and tissue-specific expression of HBsAg was detected mainly in the hepatocyte near portal area of liver and kidney . Distribution of X protein was found in the liver , kidney and brain .

HBsAg的表达具有细胞特异性（门管区周围的肝细胞）和组织特异性（肝和肾），X蛋白除在肝脏和肾脏中表达外，还可在脑组织中表达。

Conclusions Apoptosis and necrosis of VECs and SECs occur in portal area in patients with portal hypertension , which activates the blood coagulation system , promotes thrombosis and destroys hepatic microcirculation .

结论肝硬化门静脉高压症病人门管区内皮细胞和肝窦壁内皮细胞凋亡和坏死明显增加，内皮细胞的凋亡和坏死激活凝血系统，促使血栓形成，从而破坏了肝脏微循环。

Results : The liver tissue of NASH rats showed severe steatosis , and different degrees of inflammation , cell infiltration , necrosis of hepatocytes and exudation in the portal area as compared with the normal rats ;

结果：NASH模型大鼠肝组织出现了严重脂肪变和不同程度的炎症细胞浸润、肝细胞坏死、汇管区渗出；

HE staining showed necrosis of hepatocytes , thickened blood vessel wall of portal area with infiltration of lymphocytes and monocytes , hyperaemia in central vein and hepatic sinusoid , hyperplasia of Kupffer 's cell ;

苏木精伊红染色发现二氧化硫染毒可引起肝实质细胞坏死，汇管区血管壁增厚并伴有淋巴细胞和单核细胞浸润，中央静脉以及肝窦充血，库普弗细胞增生；

RESULTS : The rat liver does express both leptin mRNA and protein , its protein mainly stained at hepatic sinusoids , portal area and inflammatory regions , and its mRAN and protein expression increased as the liver fibrosis developed .

结果：大鼠肝脏表达瘦素mRNA及蛋白，瘦素蛋白阳性染色主要见于肝窦、汇管区、炎症区域，其mRNA及蛋白表达随肝纤维化发展而增多。

In the infected hepatic tissue the infiltrated cells in portal area were mainly CD4 + T lymphocytes . By the contrast , in the intralobular and perilobular necrosis area CD8 + T lymphocytes were dominant .

在感染肝组织中，汇管区浸润细胞以CD4＋T细胞为主，小叶内坏死区中以CD8＋T细胞为主，与单纯HBV感染者比较，两者差异无显著性。

IHC also showed that in most of the cases , H. pylori were located in sinus hepaticus , and in some cases , in portal area . In addition , IHC positive tissues were all shown PCR positive .

免疫组化阳性的标本PCR结果全阳性。细菌主要位于肝窦内，亦有些位于汇管区内。

The results demonstrate that MCS in the liver portal area and gastric submucosal connective tissues were stained dark brown whereas those in the gastric mucosal epithelium and in the lamina propria of the intestinal mucosa stained blue .

结果表明：肝汇管区MC呈棕褐色；胃粘膜上皮及粘膜下结缔组织内MC分别被染成蓝色和棕褐色；

The TIMP-1 , TIMP-2 related antigens in liver of immune induced model were expressed in myofibroblast and fibroblast . This was most obvious in portal area and fibrous septum . The positive signal located at cytoplasm , not in nucleus .

免疫诱导实验组肝脏中TIMP1、TIMP2相关抗原表达在肌成纤维细胞、成纤维细胞，以汇管区及纤维间隔中最明显，阳性信号位于细胞胞质中，未见细胞核表达。

Pathology morphological changes of hepatic tissue : the liver cells of the sham-operated group were mild edema . The cords of liver cells and the portal area had a few lymphocytes , and the proliferation of the small bile duct was not obvious .

肝组织病理形态学改变：假手术组肝细胞轻度水肿，肝细胞索，汇管区少量淋巴细胞浸润，小胆管增生不明显。